GABA tea

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GABA tea (also known as gabaron or gabalong ) describes a special processing technique for teas, through which the GABA ingredient is enriched in the tea leaves. This processing technique happens before the actual processing of the tea. Therefore, GABA teas can consist of different tea cultivars, cultivation regions and types of tea. As a rule, GABA teas are made from black tea , green tea (gabaron) or oolong tea (gabalong). The technique is widespread in Japan, Taiwan and China.

Manufacturing

GABA tea is based on the discovery of the Japanese tea researcher Dr. Tsushida from the 1980s. During his research on the amino acid theanine , he found that if tea leaves wither without oxygen after harvesting, the amount of GABA increases by up to 50 times. GABA occurs naturally in the tea leaf. GABA is an amino acid that has an active effect in the body and brain.

The most important ingredient in GABA tea is the amino acid γ-aminobutyric acid , or GABA for short. This amino acid occurs naturally in tea leaves. Usually with a content of 8 to 39 mg / 100 g of dry tea leaves. For most GABA teas, the concentration is around 250 mg to around 350 mg / 100 g of dry leaves.

effect

GABA has the role of a calming neurotransmitter in the central nervous system. This means that GABA ensures that the stimuli in the nervous system are slowed down or even stopped. GABA thereby promotes relaxation and can relieve anxiety. However, it is currently not exactly clear how much GABA can cross the blood-brain barrier through oral intake through tea and have an effect on the brain.

Individual evidence

  1. ↑ Tojiro Tsushida, Toshinobu Murai, Masashi Omori, Jyunko Okamoto: Production of a new type tea Containing a high level of .GAMMA.-aminobutyric acid . In: Journal of the agricultural chemical society of Japan . tape 61 , no. 7 , 1987, ISSN  0002-1407 , pp. 817–822 , doi : 10.1271 / nogeikagaku1924.61.817 ( jst.go.jp [accessed March 4, 2019]).
  2. ↑ K. Kuriyama, PY Sze: Blood-brain barrier to H3-γ-aminobutyric acid in normal and amino oxyacetic acid-treated animals . In: Neuropharmacology . tape 10 , no. 1 , January 1971, p. 103-108 .