Tachymyopathy
| Classification according to ICD-10 | |
|---|---|
| I42.9 | Cardiomyopathy, unspecified |
| ICD-10 online (WHO version 2019) | |
A Tachymyopathie ( Greek coinage ταχυμυοπάθια, tachimiopáthia , literally as "the quick [heart] muscle disease"), even Tachykardiomyopathie , tachycardia cardiomyopathy or arrhythmieinduzierte cardiomyopathy , structural heart muscle damage (is cardiomyopathy ), which on a permanently inflated heart rate (a Tachycardia : pulse over 100 beats per minute ).
Pathophysiology
The starting point is often atrial fibrillation with rapid electrical conduction in the AV node , which leads to a rapid and irregular heartbeat (a tachyarrhythmia ) with contractions of the atrium and ventricle that are no longer coordinated . Uncoordinated through the action of the heart is cardiac work ineffective, so that the muscles by so-called remodeling ( engl. Remodeling =, forming ') of the situation by z. B. tried to adjust muscle growth.
As a result of these remodeling processes, which from a certain degree impair the tissue architecture of the heart, tachymyopathy initially damages the intrinsic electrical excitation conduction system . This forwards the impulses for triggering the heart's actions from the right atrium , where the sinus node physiologically generates them, to the heart chambers, which do the main load of the actual pumping work. The damaged excitation conduction system can even favor and stabilize the causal fast frequencies in a vicious circle .
In the further course of permanent atrial fibrillation with tachyarrhythmia, there is a subsequent cardiac insufficiency (a consecutive cardiac insufficiency ) with further remodeling and manifestation of an anatomically pathologically detectable cardiomyopathy .
Therapy and relapse prevention
In the medium and long term, the main focus is on treating the underlying disease that triggers it, i.e. mostly atrial fibrillation ( see there ). Since this is often not possible, however, one tries to prevent or alleviate the effects of the underlying disease:
In order to eliminate the rapid conduction of the heart muscle excitation, drugs are administered that lower the heart rate. Usually beta blockers or verapamil or diltiazem as well as digitalis preparations are used .
In addition to lowering the heart rate, cardiac insufficiency therapy should be given.
