SLC2A10: Difference between revisions

From Wikipedia, the free encyclopedia
Content deleted Content added
m [Pu408]Tweak: title, issue. Combined duplicate references. Formatted dashes. You can use this bot yourself. Report bugs here.
→‎Further reading: added group designation to SLC navbox; removed no longer needed PBB controls and templates
Line 18: Line 18:
==Further reading==
==Further reading==
{{refbegin | 2}}
{{refbegin | 2}}
{{PBB_Further_reading
| citations =
*{{cite journal | author=Dawson PA, Mychaleckyj JC, Fossey SC, ''et al.'' |title=Sequence and functional analysis of GLUT10: a glucose transporter in the Type 2 diabetes-linked region of chromosome 20q12-13.1 |journal=Mol. Genet. Metab. |volume=74 |issue= 1–2 |pages= 186–99 |year= 2001 |pmid= 11592815 |doi= 10.1006/mgme.2001.3212 }}
*{{cite journal | author=Dawson PA, Mychaleckyj JC, Fossey SC, ''et al.'' |title=Sequence and functional analysis of GLUT10: a glucose transporter in the Type 2 diabetes-linked region of chromosome 20q12-13.1 |journal=Mol. Genet. Metab. |volume=74 |issue= 1–2 |pages= 186–99 |year= 2001 |pmid= 11592815 |doi= 10.1006/mgme.2001.3212 }}
*{{cite journal | author=Deloukas P, Matthews LH, Ashurst J, ''et al.'' |title=The DNA sequence and comparative analysis of human chromosome 20 |journal=Nature |volume=414 |issue= 6866 |pages= 865–71 |year= 2002 |pmid= 11780052 |doi= 10.1038/414865a }}
*{{cite journal | author=Deloukas P, Matthews LH, Ashurst J, ''et al.'' |title=The DNA sequence and comparative analysis of human chromosome 20 |journal=Nature |volume=414 |issue= 6866 |pages= 865–71 |year= 2002 |pmid= 11780052 |doi= 10.1038/414865a }}
Line 33: Line 31:
*{{cite journal | author=Coucke PJ, Willaert A, Wessels MW, ''et al.'' |title=Mutations in the facilitative glucose transporter GLUT10 alter angiogenesis and cause arterial tortuosity syndrome |journal=Nat. Genet. |volume=38 |issue= 4 |pages= 452–7 |year= 2006 |pmid= 16550171 |doi= 10.1038/ng1764 }}
*{{cite journal | author=Coucke PJ, Willaert A, Wessels MW, ''et al.'' |title=Mutations in the facilitative glucose transporter GLUT10 alter angiogenesis and cause arterial tortuosity syndrome |journal=Nat. Genet. |volume=38 |issue= 4 |pages= 452–7 |year= 2006 |pmid= 16550171 |doi= 10.1038/ng1764 }}
*{{cite journal | author=Lin WH, Chuang LM, Chen CH, ''et al.'' |title=Association study of genetic polymorphisms of SLC2A10 gene and type 2 diabetes in the Taiwanese population |journal=Diabetologia |volume=49 |issue= 6 |pages= 1214–21 |year= 2006 |pmid= 16586067 |doi= 10.1007/s00125-006-0218-3 }}
*{{cite journal | author=Lin WH, Chuang LM, Chen CH, ''et al.'' |title=Association study of genetic polymorphisms of SLC2A10 gene and type 2 diabetes in the Taiwanese population |journal=Diabetologia |volume=49 |issue= 6 |pages= 1214–21 |year= 2006 |pmid= 16586067 |doi= 10.1007/s00125-006-0218-3 }}
}}
{{refend}}
{{refend}}


{{membrane-protein-stub}}
{{membrane-protein-stub}}
{{NLM content}}
{{NLM content}}
{{Solute carrier family|bg|bg0}}
{{Membrane transport proteins}}
[[Category:Solute carrier family]]
[[Category:Solute carrier family]]

<!-- The PBB_Controls template provides controls for Protein Box Bot, please see Template:PBB_Controls for details. -->
{{PBB_Controls
| update_page = yes
| require_manual_inspection = no
| update_protein_box = yes
| update_summary = yes
| update_citations = yes
}}

Revision as of 09:17, 25 November 2012

Template:PBB Solute carrier family 2, facilitated glucose transporter member 10 is a protein that in humans is encoded by the SLC2A10 gene.[1][2]

Template:PBB Summary

See also

References

  1. ^ McVie-Wylie AJ, Lamson DR, Chen YT (2001). "Molecular cloning of a novel member of the GLUT family of transporters, SLC2a10 (GLUT10), localized on chromosome 20q13.1: a candidate gene for NIDDM susceptibility". Genomics. 72 (1): 113–7. doi:10.1006/geno.2000.6457. PMID 11247674. {{cite journal}}: Unknown parameter |month= ignored (help)CS1 maint: multiple names: authors list (link)
  2. ^ "Entrez Gene: SLC2A10 solute carrier family 2 (facilitated glucose transporter), member 10".

Further reading

This article incorporates text from the United States National Library of Medicine, which is in the public domain.