Hyperkinetic heart syndrome
| Classification according to ICD-10 | |
|---|---|
| I51.8 | Other imprecisely specified heart diseases Hyperkinetic heart syndrome |
| ICD-10 online (WHO version 2019) | |
The hyperkinetic heart syndrome ( English : hyperkinetic heart syndrome ) describes a complex of symptoms consisting of a rapid pulse, high blood pressure, decreased performance and unsystematic dizziness. It is counted among the hyperdynamic circulatory disorders in the context of functional heart problems and should be a diagnosis of exclusion . The syndrome was first described in 1960.
Clinical manifestations
The hyperkinetic heart syndrome occurs mainly in younger people, here mainly in women, and is noticeable through a combination of various symptoms . There is a rapid pulse ( tachycardia ) at rest and an excessive increase in the heart rate during exercise. Furthermore, there is usually a predominantly systolic increased blood pressure . Those affected describe reduced physical resilience and unspecific abnormal sensations in the heart area as well as dizziness .
causes
In addition to a pronounced lack of physical fitness due to lack of exercise / training , an increased sensitivity of receptors for the hormones of the sympathetic nervous system (increased sensitivity of the β-1 receptors ) has been discussed in the past . However, neither an increased activity of the sympathetic nervous system or its receptors nor a decreased activity of the parasympathetic nervous system could be demonstrated, so that today one assumes a central nervous malfunction. Mental causes in the sense of anxiety disorders are often accompanying.
Investigation methods
Before diagnosing hyperkinetic heart syndrome, other causes should be ruled out. To this end, a detailed should anamnesis collected and in addition to a physical examination should also resting ECG , stress ECG , Holter , long-term blood pressure monitoring , echocardiography , laboratory tests ( blood count , sodium , potassium , creatinine , CRP , TSH , possibly with urine on catecholamines ) to be created. In addition, an electrophysiological examination may be necessary.
- endocrine (hormonal) diseases such as overactive thyroid gland ( hyperthyroidism ) and adrenal glands ( pheochromocytoma ),
- Use of drugs or stimulants such as caffeine , ecstasy , cocaine , black tea ,
- Overdosage of some nasal or asthma sprays ( β-1 mimetics ) and
- Arrhythmias such as AV nodal reentry tachycardia
into consideration.
A largely similar disease with a detectable change in the mitral valve of the heart is the mitral valve prolapse syndrome .
treatment
Initially, the focus is on educating people about the benign nature of the disease. The administration of low-dose beta-blockers can break the vicious circle of fear and a rapid pulse. An increase in physical performance through training is desirable, heart-stimulating substances such as caffeine should be avoided. Learning relaxation techniques is generally helpful. If necessary, the patient should seek psychotherapy . In this context , a decision can be made about the need to administer psychotropic drugs .
Individual evidence
- ↑ a b c d Gerhard Steinbeck, Gustav Paumgartner (Ed.): Therapy for internal diseases . 11th edition. Springer, Heidelberg 2005, ISBN 3-540-23750-X , p. 227 f .
- ↑ N. Brachfeld, R. Gorlin: Idiopathic hyperkinetic state: a new clinical syndrome . In: British Heart Journal . tape June 22 , 1960, p. 353-360 , PMID 13803668 , PMC 1017666 (free full text).
- ↑ a b C. Vallbracht, M. Kaltenbach (Ed.): Cardiovascular compact . 1st edition. Steinkopf, Darmstadt 2006, ISBN 3-7985-1495-X , p. 439-441 .
- ^ Matthias Lohr, Bernhard K. Keppler: Internal medicine. Compendium for studies and clinics. Elsevier, Munich 2005, p. 87.
