UCP2: Difference between revisions
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*{{cite journal | author=Arsenijevic D, Onuma H, ''et al.'' |title=Disruption of the uncoupling protein-2 gene in mice reveals a role in immunity and reactive oxygen species production. |journal=Nature Genetics |volume=26 |issue= Dec |pages= 435-9 |year= 2000 }} |
*{{cite journal | author=Arsenijevic D, Onuma H, ''et al.'' |title=Disruption of the uncoupling protein-2 gene in mice reveals a role in immunity and reactive oxygen species production. |journal=Nature Genetics |volume=26 |issue= Dec |pages= 435-9 |year= 2000 }} |
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*{{cite journal | author=Bai Y, Onuma H, Bai X, ''et al.'' |title=Persistent nuclear factor-KB activation in Ucp2-/- mice leads to enhanced nitric oxide and inflammatory cytokine production. |journal=J Biol Chem |volume=280 |issue= May 13 |pages= 19062-9 |year= 2005 }} |
*{{cite journal | author=Bai Y, Onuma H, Bai X, ''et al.'' |title=Persistent nuclear factor-KB activation in Ucp2-/- mice leads to enhanced nitric oxide and inflammatory cytokine production. |journal=J Biol Chem |volume=280 |issue= May 13 |pages= 19062-9 |year= 2005 }} |
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Revision as of 16:49, 24 January 2008
Uncoupling protein 2 (mitochondrial, proton carrier), also known as UCP2, is a human gene.
References
- ^ a b c GRCh38: Ensembl release 89: ENSG00000175567 – Ensembl, May 2017
- ^ a b c GRCm38: Ensembl release 89: ENSMUSG00000033685 – Ensembl, May 2017
- ^ "Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
- ^ "Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
Further reading
- Arsenijevic D, Onuma H; et al. (2000). "Disruption of the uncoupling protein-2 gene in mice reveals a role in immunity and reactive oxygen species production". Nature Genetics. 26 (Dec): 435–9.
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(help) - Bai Y, Onuma H, Bai X; et al. (2005). "Persistent nuclear factor-KB activation in Ucp2-/- mice leads to enhanced nitric oxide and inflammatory cytokine production". J Biol Chem. 280 (May 13): 19062–9.
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This article incorporates text from the United States National Library of Medicine, which is in the public domain.